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Secondary outcomes The secondary outcomes are as follows: type 2 diabetes, glucose, HbA1c, insulin, lipid profile (LDL-cholesterol, HDL-cholesterol, triglycerides and apolipoprotein B), systolic blood pressure, diastolic blood pressure, and body mass index (BMI), which are well-established risk factors for CVD in clinical practice, or recently identified to be related to CVD in MR, such as apolipoprotein B [30, 31]

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Correspondingly, left ventricular internal dimensions and end volume in the HUA group were decreased compare with those in the blank group, but LC intervention ameliorated the HUA-associated decrease in internal dimensions and end volume in diastolic-stage rather than systolic-stage, as did the urate-lowering agent AP, while TMZ did not show significant improvement (Figures 6FI)

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