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Because of an elevated risk of cardiovascular morbidity and mortality in NASH patients prior to advanced fibrosis, as well as hepatic decompensation, HCC, and/or liver failure, therapeutic options must address the complex pathophysiology of this disease
The GHR is a challenging target in this regard in that it lacks a kinase domain and the ligand binding surface involves a large, relatively featureless protein-protein interface
(1) WithB Vitamins to help convert food to fuel (2) Not a replacement for heart medications *This statement has not been evaluated by the Food and Drug Administration
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