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Table 2 details the phases 2, 3, and 4 clinical trials that have assessed, or are assessing, anti-A therapies in Alzheimers disease

This protective effect is attributed to mechanisms such as the augmentation of leaf turgor, elevation of osmolyte levels, and mitigation of oxidative damage through the stimulation of antioxidant activities (Sita and Kumar 2020)

This selectivity profile made it particularly attractive for clinical development and is the primary reason it became the most widely used GHRP in the peptide therapy space

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