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In different in vitro models, HPE gave protection by suppressing lipid peroxidation, nitric oxide, hydroxyl radical and superoxide anion generation 63
Studied in models involving cellular aging pathways, circadian rhythm biology, pineal signaling, telomere-associated research, and stress-response systems
[32] to be caused by crosslinking between cysteine residues located in the 6 helices of Bak proteins
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