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10.1038/srep23204 16 AtkinJ
Polyphenols can suppress the expression and activity of key pro-inflammatory mediators, such as nuclear factor-B (NF-B), a transcription factor vital to the regulation of the inflammatory response, as shown in Table 2 (269, 270)
One interpretation that is often cited regarding such case reports is that these were examples of a dysmyelination resulting from a defined gene defect or an acquired/sporadic condition, with coincidental MS (Pfeffer et al., 2013
When used in combination, these compounds support exploration of convergent signalling responses, matrix-associated processes, and peptidemetal interactions under controlled laboratory conditions
