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Once synthesised, GSH is exported from the cytosol and actively transported into mitochondria, where it maintains mitochondrial redox balance and protects mitochondrial DNA from oxidative damage
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The HbA1c reduction, as with other antihyperglycaemic agents depends largely on the patient population studied, the baseline glycaemic situation at the beginning of the observation and the concomitant therapy, including life-style intervention (12, 19, 21, 22)
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