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One study demonstrated that semaglutide inhibits the transformation of astrocytes into a neurotoxic A1 phenotype, a process that contributes to BBB breakdown and neuronal damage following ischemia (Tang et al., 2022)
Residual stiffness is common but should not be severe
Genome-wide association analyses have detected critical polymorphisms in genes regulating hepatic lipid handling, including patatin-like phospholipase domain-containing 3 (PNPLA3) and transmembrane 6 superfamily member 2 (TM6SF2), which promote intrahepatic triglyceride accumulation by impairing lipid remodeling or very-low-density lipoprotein (VLDL) secretion [16,17]
1A) [8]
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