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Transgenic mice that develop thyroid-specific autoantigens in a spontaneous manner have, therefore, been very useful and provide a good platform for studying immunological mechanisms of the disease as reported by Merrill and Pandiyan (2018) (Merrill and Pandiyan 2018)

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Compared to CCR10 - ECs, CCR10 + ECs exhibited robust proangiogenic capacity, which was induced by CCL28 through CCR10/ERK/SOX5 positive feedback signaling