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Finally, we used to model to show how the observed metabolic profile of Down syndrome could arise from trisomy 21 and moderate oxidative stress and how the observed metabolic profile of autism patients could arise from high oxidative stress and increased adenosine concentration
Roles of the glutathione- and thioredoxin-dependent reduction system in the Escherichia coli and Saccharomyces cerevisiae responses to oxidative stress
Our findings of decreased NRF2 levels seem to contradict previous studies suggesting an increased NRF2 response in Down syndrome
However, some studies do discuss the related risks with fluctuations in IGF1 and its binding proteins
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