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In particular, increased plasma insulin concentrations stimulate the gonadotropin releasing hormone (GnRH) pulsatility and potentiate the LH-dependent effect on the ovarian theca cells, leading to the enhanced synthesis of androgens and to the arrest of pre-antral follicle development [13]
Sermorelin stimulates the pituitary gland to produce GH, while GHRP-6 amplifies this effect by binding to specific receptors that trigger additional GH secretion
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