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second, cisplatin can directly inhibit the physiological function of human iron regulatory protein 2 by covalently binding to its cysteine residue sites (Cys512 and Cys516), disrupting the homeostasis of systemic iron metabolism, causing persistent intracellular iron deficiency, and further aggravating refractory anemia (Miyazawa et al
Sarah Chen, PharmD, BCPS, with 14 years of experience in metabolic medications and clinical pharmacy practice
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In its main 56-week clinical trial, patients on the highest dose lost an average of 8 percent of their starting body weight
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