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1e and the rate constants from Table 1 (Fig
Initially, we delve deeply into the pathophysiological mechanisms underlying AMS, elaborating extensively on core aspects such as adaptive responses to hypoxia, hemodynamic alterations, increased blood viscosity, and the resultant spectrum of tissue damage and complications
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Through shared precursors, metabolic interactions, and functional collaborations, MLT and intestinal Trp metabolites constitute a classic hostmicrobial co-metabolic network ( Allobaculum and Parasutterella
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