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Further mechanistic studies showed that NNMTi increases intracellular NAD levels and activates the SIRT1 pathway by inhibiting NNMT, thereby suppressing the transcription of adipogenic genes and preventing the maturation of preadipocytes.This research provides a new perspective for understanding the molecular mechanism of adipocyte differentiation, confirms the key role of NNMTi in regulating adipogenesis, and lays a foundation for subsequent obesity intervention studies

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Research also highlights BPC-157s role in skeletal muscle healing, cell membrane signaling (including caveolin-1 and caveolae), and activation of the FAK-paxillin pathway, which is important for cellular adhesion and migration

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