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The overproduction of lactate would thus be secreted into the circulation through MCT4 and preferentially bind to GPR81 on endothelial cells, to reduce endothelial VE-Cadherin expression and increase vascular permeability through different signaling
Acknowledgements This work is supported in part by NIH grants AA022057 (VV), AA021724 (VV)
have shown that DENV-2 infection alters oxidative stress by increasing the levels of malondialdehyde (MDA), the end product of lipid peroxidation, and the oxidized glutathione/GSH ratio 126
Animals were singly housed before the experiment and fasted for 2 h
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