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Background and Summary of Retrospective Studies Homocystinuria caused by congenital CBS and MTHFR defects or by genetic alteration of cobalamin metabolism is characterized by clinical problems including frequent arterial and venous thromboembolism and premature atherosclerosis (McCully, 1983
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Gene Regulation: Downregulates senescence markers like p16 (INK4a) and p21 (Waf1), delaying cellular aging
If blood work shows healthy B12 levels and you already supplement B12 orally, choosing niacinamide over B12 as your compound additive avoids redundancy
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