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Mechanism appears multifactorial: upregulation of nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), and synaptic plasticity proteins like synaptophysin
Both excitatory and inhibitory synaptic genes show decreased expression with age and are positively correlated with BDNF and NTRK2 expression and negatively correlated with dominant-negative truncated NTRK2 level
On the other hand, the treatment policity estimand was used to evaluate the treatment effect for all randomized participants regardless of whether the trial was discontinued early or rescue medicien was used [2]
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