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CCL11 enhances excitotoxic neuronal death by producing reactive oxygen species in microglia
The result: telomerase catalytic subunit expression was reactivated, enzyme activity increased, and telomere lengthening occurred, effectively extending the cells proliferative capacity from ~34 to over 44 passages [1]
For the maintenance treatment of vitamin B12 deficiency, the usual dose is 60-100mcg SC/IM, given once per month
Supports genomic stability research: Telomere shortening can lead to chromosomal instability and altered gene expression
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