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Deletion of PRX1 eliminated H 2 O 2 induced oxidation of matrix glutathione and elicited a transcriptional response that increased levels of the cytosolic catalase, Ctt1, showing that cells can recognize, and respond to, impaired matrix redox homeostasis

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However, the evidence in humans remains close to null, and the evidence that does exist has many important limitations, including that none were conducted in overweight patients