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Similarly, -synuclein aggregation can suppress system Xc activity, leading to glutathione depletion, whereas membrane damage caused by ferroptosis may in turn facilitate the intercellular spread of -synuclein
In addition, while individuals with renal insufficiency are at higher risk of developing CAD compared to those with normal kidney function, that risk was not found to be greater with low rather than normal selenium concentrations in serum (98 g/L vs
Arch Immunol Ther Exp
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