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Kanoe Namahoe, 54, was diagnosed with type 2 diabetes in January and has high blood pressure
NO synthase (eNOS) phosphorylation Furthermore, exenatide reversed homocysteine-induced endothelial dysfunction by decreasing sICAM-1 and reactive oxygen species (ROS) levels and upregulating NO production and eNOS phosphorylation which further contributed the improvement of coronary endothelial function in diabetic patients [58]
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8f), and this correlates with our reported increase in mitochondrial mass, ATP production and mTOR activity (Figs
