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It has been proposed that depletion of hepatic GSH, particularly mitochondrial GSH, is one of the early changes associated with chronic alcohol consumption and is a critical contributor to ALD pathogenesis 11
Amorim, G
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Resistance after CF-based NAC may be driven predominantly by the selection or enrichment of pre-existing resistant subclones or cell states, rather than by extensive therapy-induced reprogramming
In addition, BAS sevelamer suppresses the acute bile acid-induced GLP-1 secretion in patients with T2DM
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