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If your research requires absolutely clean IGF-1R-specific signalling without any insulin receptor cross-reactivity, DES is theoretically superior
A.Ingelmo-TorresM.FergusonC.EnrichC.et al (2005)
[3] Potency: IC: ~1.01.2 M for NNMT inhibition [1] EC: 2.3 1.1 M (reduction of intracellular 1-MNA levels) [1] Lipogenesis Inhibition: 30 M reduced lipogenesis by 50%
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