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Besides triggering a sequence of events that cause toxic outcomes, exposure to xenobiotics leads to induced oxidative stress, generating an over production of reactive oxygen species (ROS) [49] and consequently triggering oxidative damage to macromolecules such as proteins, lipids, and nucleic acids [50, 51]

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After binding to GLP-1 receptors in pancreatic beta cells, GLP-1 RAs exert their glucose-lowering effect by stimulating glucose-dependent insulin release