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Furthermore, when leaked into the extracellular microenvironment, the intermediate metabolites of melanogenesis, which are also immunosuppressive, may promote evasion of immune responses by the initially transformed melanocyte precursors and their offspring melanoma cells, increasing the risk of melanomagenesis and melanoma growth [60]

These modifications enhance its stability and prolong its interaction with receptors in experimental systems, distinguishing it from native GLP-1, which is rapidly degraded in the body

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