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Their framework helps explain something my patients describe all the time: the medication turns down the internal, homeostatic drive to eat, the "I'm running low on fuel" signal, but external and emotional cues run on partly separate circuitry
This triggers a dual effect on: Glucose-Dependent Secretion: The rise in cAMP activates Protein Kinase A (PKA) and Epac (Exchange Protein directly activated by cAMP)
doi: 10.1016/j.tem.2023.10.005 10 TangRZhongTLeiKLinXLiX
The slowed gastric emptying at 1.0 mg is significant enough that food sits in the stomach much longer than usual, creating a persistent fullness and distension that some find quite uncomfortable
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