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Discover the full paper here: doi.org/10.1113/JP290260 #Electrophysiology #PatchClamp #ePatch #IonChannels #NeuropathicPain #HCN2Channels #HCN2 #cAMP #Neurobiology #Pharmacology #DrugDiscovery #CellSignaling #Neuropharmacology #PainResearch #DRGNeurons #DRG #TRIP8bnano #Nociceptors #Hypersensitivity #Allodynia #Hyperalgesia #SNL To view or add a comment, sign in I am honoured to be sharing the stage with Prof
Gpxs exist in multiple isoforms, with different cellular localization and different substrate specificity ( 2 O 2 , Prxs seem to be more active, given their higher intracellular concentration and rate constant ( GSH, exploiting the redox-active thiol residue (-SH) of cysteine, exerts its antioxidant function mainly via Gpxs-mediated reactions, which result in peroxide buffering with simultaneous oxidation of GSH to GSSG ( Besides the neutralization of free radicals produced in phase 1 liver metabolism of chemical toxins, GSH also participates in the protection from the resulting electrophilic substrates through the intervention of glutathione-S-transferases (GSTs)
W., Jarrad, A
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