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These findings highlight that GLP-1-RA such as Ex-4 can promote hMSC proliferation, while native GLP-1 forms and Liraglutide predominantly exert inhibitory effects on proliferation and glucose uptake, with varying results under different conditions, highlighting their context-dependent role in hMSC behavior and metabolic regulation

BPC 157 was also shown to time-dependently activate the VEGFR2-Akt-eNOS signalling pathway

Ferroptosis in B cells is controlled by similar signaling mechanisms (81)

The treatment policy (secondary) estimand evaluated the treatment effect for all randomised participants regardless of trial product discontinuation or use of rescue medication