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By eliminating senescent cells in preclinical systems, FOXO4-DRI indirectly reduces the source of SASP factors
Damage to endothelial cells results in heightened secretion of the vasoconstrictor endothelin and diminished secretion of the vasodilator nitric oxide, causing sustained renal vasoconstriction, impaired renal perfusion, and aggravated renal injury [83]
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