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Oral N-acetylcysteine rescues lethality of hepatocyte-specific Gclc-knockout mice, providing a model for hepatic cirrhosis

Relative to IV administration, PO administration results in poor bioavailability of ketamine (9-12%), inverted pharmacologically active norketamine and hydroxynorketamine to ketamine metabolite-to-parent compound ratios (being higher for ketamine PO and lower for ketamine IV), and limited to complete absence of PD effects relative to IV administration

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