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Endothelial cell produced NO can also interfere with leukocyte adhesion to the endothelium thus, further contributing to a reduction in the potential for atherosclerosis
( H ) GIP, GLP-1, insulin, and glucose levels in mice treated with the ghrelin receptor antagonist d-Lys3 before glucose administration
Previous studies suggested that TRPM5 activation and the subsequent increases in Na + -influx play a crucial role in GLP-1-induced increases in beta cell electrical activity [28]
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