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Likewise, considering the beneficial effect of the deletion of the Nogo Receptor 1 (NgR1) gene, a sequential combination of Nogo-A suppression (by anti-Nogo-A antibody treatment) and treadmill rehabilitative training was examined [25]

This mechanistic pathway requires the activity of iron-dependent enzymes such as lipoxygenases, ferroptosis suppressor protein 1, glutathione peroxidase 4, dihydroorotate dehydrogenase, and lysosomal iron release from ferritin stores

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