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Kupfer and Regier 2011
GHK-Cu and Cellular Senescence: Downstream Oxidative Mechanism In aged fibroblasts identified by high SA--gal activity, GHK-Cu reduces SA--gal+ cells from 48% to 28% (41%), p16INK4a mRNA 2832%, and p21WAF1 2226% with 7-day treatment indicating partial reversal of the senescent phenotype
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Improper use of the devices or inexpensive, non-FDA-cleared technologies may result in bad outcomes, such as pigmentation issues or scarring
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