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Potentiation of glucose-induced insulin secretion by GIP is diminished in chronic hyperglycaemic states due at least in part to a decrease in the Gs subtype of the GIP receptor in pancreatic beta cells, and further reasons for concern over GIP as a treatment for type 2 diabetes are that it increases glucagon secretion and adipose deposition [49]
At position 26, a C18 fatty diacid chain is attached to the lysine residue through a linker molecule
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