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Lymphocyte depletion in lymphoid organs due to APAP has already been observed, but at high doses and it was associated with hepatotoxicity 45,51

Interestingly, in both the absence and presence of IL-1, GS dose-dependently inhibited PGE 2 production, and significance was reached at 0.2 mM for the endogenous and at 1 mM for IL-1-induced production of PGE 2 (Figure 1)