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4.4 Neuroinflammation The neurotoxic environment created by synthetic cathinones activates microglia and astrocytes, leading to the release of proinflammatory cytokines, such as IL-1 and TNF-
Kolbrink, B
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Following luteolin treatment modulated airway bronchoconstriction and bronchial hyperreaction and immunologically reduced OVA-specific IgE levels, IL-4, and IL-5 levels, and increased IFN- levels in the BALF [158]
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