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Activation of the NLRP3 inflammasome is often triggered by environmental allergens, leading to the cleavage of pro-IL1 into its active form via caspase-1, which subsequently promotes a pro-inflammatory milieu conducive to Th17 cell differentiation and neutrophil recruitment [150, 151]

In this regard, the intranasal route is a promising delivery method to obtain greater efficacy in maintaining optimal drug concentrations in the brain compared to parenteral injections [17-20]

GHK-CU This Tripeptide has been shown to enhance elasticity, reduce inflammation, and protect against oxidative damage