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Deacon CF, Johnsen AH, Holst JJ (1995) Degradation of glucagon-like peptide-1 by human plasma in vitro yields an N-terminally truncated peptide that is a major endogenous metabolite in vivo
A clean RS divergence into VWAP/ORH/ORL often gives tight-risk rotations
Recent work has also identified the source of DPP-4 critical to GLP-1 and GIP degradation as being endothelial cells, rather than enteric epithelial or haematopoietic cells [19]
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