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In animal models, it has been shown preclinically to upregulate growth hormone receptors in tendon fibroblasts, stimulate angiogenesis via the VEGFR2/Akt-eNOS pathway, and modulate nitric oxide signaling, mechanisms that, if they translate to humans, could support tissue repair in a hormonal environment where estrogen-dependent healing capacity is declining
The anti-inflammatory properties of both peptides may contribute to joint health maintenance through reduction of inflammatory mediators that contribute to cartilage breakdown
Q: Is BPC 157 approved to treat injuries or inflammation
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