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Metformin treatment decreased the abundance of species of Bacteroides fragilis and its bile salt hydrolase activity in the intestines of individuals with T2D, increasing the levels of the bile acid glycoursodeoxycholic acid to inhibit intestinal FXR signaling (47)
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Combined with differential metabolite data, complete causal chains were constructed, and genes corresponding to target eQTLs were selected as complete mediators
The following year, the FDA approved higher doses of tirzepatide to treat obesity
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