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Non-neural adrenergic and cholinergic signalling pathways are expressed by melanocytes
Am J Physiol (1987) 252(1 Pt 2):R1605
(54) found that administering GIPR antagonist to healthy humans reduced postprandial insulin secretion and increased glucose excursions more than administering GLP-1R antagonist (GLP-1RA), indicating that endogenous GIP may play a more significant role in stimulating insulin secretion and reducing postprandial plasma glucose excursions compared to endogenous GLP-1
Tissue repair Studied for the combined effect of BPC-157 and TB-500 on wound healing and regeneration
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