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For example, exendin-4 (Ex-4), a GLP-1R agonist, increases excitatory synaptic inputs to the anorexigenic corticotropin-releasing hormone (CRH) neurons in the paraventricular nucleus of the hypothalamus (PVH) [2,3]

It targets and inhibits the activity of an enzyme known as NNMT, which has been linked to a slowdown in metabolism and a reduction in energy production

Research on GLP-1 therapies shows that brief interruptions of 12 weeks don't erase the weight loss achieved or the pathway changes established in your nervous system

GLP-1 Multiple studies in rodents have established the role of GLP-1 in bone metabolism