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For researchers in younger demographics, the same protocols apply, though the necessity of telomerase activation in individuals with adequate telomere length remains an open question

Remodeling of whole-body lipid metabolism and a diabetic-like phenotype caused by loss of CDK1 and hepatocyte division

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Importantly, these findings demonstrate that while the depletion of glutamate by GCLC is a metabolic vulnerability under nutrient replete conditions, we find that it confers an advantage under cysteine starved conditions