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In individuals with T2DM, chronic hyperglycemia is known to downregulate GIPR expression and impair GIP-stimulated insulinotropic activity, thereby limiting the efficacy of GIP monotherapy [21]
Its safety, efficacy, and pharmacological effects in humans have not been established
Moreover, in MAR709 experiments, the attachment of exenatide (2939) has a limited positive effect on GLP-1 activity (EC 50 : 0.022 nM0.020 nM)
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